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: 1950))Ĭited: 14 times View full text PDF listing > J Immunol (Journal of immunology (Baltimore, Md. In juvenile idiopathic arthritis (JIA) patients, we have previously reported that atypical CD25(+)FOXP3(-) Treg-like cells uniquely populate the inflamed site. Synovial Regulatory T Cells Occupy a Discrete TCR Niche in Human Arthritis and Require Local Signals To Stabilize FOXP3 Protein Expression.ĭavid Bending, Eirini Giannakopoulou, Hannah Lom, Lucy R Wedderburn,Īlthough there is great interest in harnessing the immunosuppressive potential of FOXP3(+) regulatory T cells (Tregs) for treating autoimmunity, a sizeable knowledge gap exists regarding Treg fate in human disease. Read more >Īrthritis Rheumatol (Arthritis & rheumatology (Hoboken, N.J.))Ĭited: 8 times View full text PDF listing > To date, analysis of juvenile idiopathic arthritis (JIA) immune pathology has concentrated on the contribution of CD4+ T cells we have previously identified an. ObjectiveEvidence suggests that aberrant function of innate lymphoid cells (ILCs), whose functional and transcriptional profiles overlap with those of Th cell subsets, contributes to immune-mediated pathologies. Innate Lymphoid Cells and T Cells Contribute to the Interleukin-17A Signature Detected in the Synovial Fluid of Patients With Juvenile Idiopathic Arthritis.Įlizabeth C Rosser, Hannah Lom, David Bending, Chantal L Duurland, Mona Bajaj-Elliott, Lucy R Wedderburn,